Appearance : Almost white, powder
Solubility: Soluble in water at 54 Deg C
Melting Point 48-50 Deg C
Loss on Drying 0.08% ( max limit Not more than 0.2%)
Assay By GLC  99.12%
Phenol Content by GLC 0.009% ( Max allowable 0.05%)
Colour Absorption :Complies

 Toxicity Data

Human health risk

Animal data indicate that TPP has low toxicity. It produces no irritant effects on animal skin. Despite an early report to the contrary, TPP is not considered neurotoxic in animals or man. TPP is not mutagenic. The available data indicate no hazards to man.

No evidence that TPP causes delayed neurotoxicity has been found in animal experiments. No adequate data on the effects of TPP on reproduction are available. Contact dermatitis due to TPP has been described.

Exposure

Exposure of the general population to TPP through various environmental media is likely. TPP has often been detected in urban air, although the levels are low.

The presence of TPP and other organophosphorus compounds in the indoor environment has recently been reported

Environmental fate and levels

Triaryl phosphates (including TPP) enter into the aquatic environment mainly via hydraulic fluid leakage as well as by leaching and volatilisation from plastics, and to a minor extent, from manufacturing processes. Triphenyl phosphate is rapidly adsorbed on sediments. Its biodegradation is rapid. The bioconcentration factors measured for several species of fish range from 6-18,900 and the depuration half-life ranges from 1.2 to 49.6 hours.

Maximum environmental levels reported are 23.2 ng/m3 in air, 7,900 ng/l in river water, 4,000 ng/g in sediment, and 600 ng/g in fish.

Effects on organisms in the environment

The growth of algae is completely inhibited at TPP concentrations of 1 mg/l or more, but is stimulated at lower concentrations.

TPP is the most acute toxic of the various triaryl phosphates to fish, shrimps and daphnids. The acute toxicity index of TPP for fish (96 h LC50) ranges from 0.36 mg/l in rainbow trout to 290 mg/l in bluegills. Sublethal effects on fish include morphological abnormalities such as congestion, degeneration, and haemorrhage from the smaller blood vessels and behavioural abnormalities. The immobility of fish exposed to 0.21-0.29 mg per litre completely disappeared within 7 days, when the fish were transferred to clean water